herbal medicine schools - Hypertension relationships and Menopause ; Gender plays an important role on blood pressure. Premenopausal women have lower blood pressure than men of the same age.

Compared with premenopausal women, postmenopausal women had higher blood pressure. This suggests that the ovarian hormones may modulate blood pressure.

It was reported that both the systolic blood pressure and diastolic closely related to menopausal age, BMI (Body Mass Index), changes in hormone therapy, and pulse.

To find out more about the relationship between menopause with hypertension there is research done by Megan Coylewright and colleagues. The results found that women in menopause higher blood pressure than premenopausal women.

Women in menopause were found to have a systolic blood pressure greater than men with the same BMI and age. While systolic blood pressure increased 5mm/Hg in five years.

The increase in systolic blood pressure showed a decrease in arterial adjustment. The relationship between blood pressure and hormone replacement therapy (HRT) found that those taking hormone replacement therapy had lower blood pressure slightly lower than those who did not use HRT. Once again this shows that there is a relationship between a hormone produced by the ovaries with the woman's blood pressure. It is then examined as a cause of hypertension in postmenopausal women.

Research on hypertension in postmenopausal women and its association with female hormones also reveals the role and how hormones can affect blood pressure. Menopause is associated with a reduction in estradiol and a decrease in the ratio of estrogen and testosterone.

This results in endothelial dysfunction and increased BMI leads to an increase in the activation of the sympathetic nerves that often occurs in women who experience menopause. Activation of the sympathetic nervous stimulants would eject renin and angiotensin II.

Dysfunction endhotelial eventually increase sensitivity to salt and increase endhotelin. Not only that, the increase in angiotensin and endhotelin can cause oxidative stress that eventually leads to hypertension or high blood pressure.

A similar study revealed that estrogen can reverse the progression of hypertension. Just by adding a low-dose estrogen in hypertensive rats, this hormone is able to prevent the development of high blood pressure to heart failure right.

Preventing the development does not occur in mice that were not given estrogen. After the administration of low-dose estrogen for 10 days suspended, apparently preventing the development of high blood pressure to heart disease still take up to 12 days.

Is elevated blood pressure, decrease, or remain with regard to estrogen therapy is highly dependent on three factors: the type of estrogen, estrogen dose, and how blood pressure is monitored.

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